Assessment of acute pulmonary vascular reactivity in portopulmonary hypertension

Giovanni L. Ricci, Maria Teresa Melgosa, Felip Burgos, José Luis Valera, Sandra Pizarro, Josep Roca, Roberto Rodriguez‐Roisin, Joan Albert Barberà – 29 October 2007 – The role of acute pulmonary vasodilator testing in portopulmonary hypertension (PoPH), a current contraindication for orthotopic liver transplantation (OLT), has not been thoroughly elucidated.

Streptococcus salivarius bacteremia and spontaneous bacterial peritonitis in liver transplantation candidates

Manjushree Gautam, Kapil B. Chopra, David D. Douglas, Rebecca A. Stewart, Shimon Kusne – 29 October 2007 – Bacterial infections are a serious complication of end‐stage liver disease (ESLD) that occurs in 20% to 60% of patients. We retrospectively reviewed medical records of patients with ESLD who were identified by our microbiology laboratory as having Streptococcus salivarius bacteremia. Of 592 patients listed for transplantation between January 1998 and January 2006, 9 (1.5%) had 10 episodes of S salivarius bacteremia.

Outcomes of donor evaluation in adult‐to‐adult living donor liver transplantation

James F. Trotter, Karen A. Wisniewski, Norah A. Terrault, James E. Everhart, Milan Kinkhabwala, Robert M. Weinrieb, Jeffrey H. Fair, Robert A. Fisher, Alan J. Koffron, Sammy Saab, Robert M. Merion, A2ALL Study Group – 29 October 2007 – The purpose of donor evaluation for adult‐to‐adult living donor liver transplantation (LDLT) is to discover medical conditions that could increase the donor postoperative risk of complications and to determine whether the donor can yield a suitable graft for the recipient.

Decreased white matter lesion volume and improved cognitive function after liver transplantation

Alex Rovira, Beatriz Mínguez, F. Xavier Aymerich, Carlos Jacas, Elena Huerga, Juan Córdoba, Juli Alonso – 29 October 2007 – Focal T2‐weighted white matter lesions (WML) on brain magnetic resonance imaging (MRI), mimicking those seen in cerebrovascular small‐vessel disease described in patients with persistent hepatic encephalopathy, decreased in volume with the improvement of hepatic encephalopathy. This outcome has been interpreted as a decrease in the edema that it is proposed to be involved in the pathogenesis of hepatic encephalopathy.

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