Persistent hepatitis C virus–associated cryoglobulinemic vasculitis following virus eradication after direct‐acting antiviral therapy
Marina Artemova, Dzhamal Abdurakhmanov, Tatiana Ignatova, Nikolay Mukhin – 10 December 2016
Marina Artemova, Dzhamal Abdurakhmanov, Tatiana Ignatova, Nikolay Mukhin – 10 December 2016
Tian Yang, Han Zhang, Jun Han, Meng‐Chao Wu, Feng Shen – 10 December 2016
Ruben Hernaez, Hashem B. El‐Serag – 10 December 2016
Yu Tahara, Shigenobu Shibata – 10 December 2016
Javier Martínez, Luis Téllez, Agustin Albillos – 10 December 2016
Anna Linda Zignego, Laura Gragnani, Marcella Visentini, Milvia Casato – 10 December 2016
Suk Kyun Hong, Kwang‐Woong Lee, Hyo‐Sin Kim, Kyung Chul Yoon, Sung‐Woo Ahn, Jin Yong Choi, Hyeyoung Kim, Nam‐Joon Yi, Kyung‐Suk Suh – 9 December 2016
Margaret V. Ragni, Abhinav Humar, Peter G. Stock, Emily A. Blumberg, Bijan Eghtesad, John J. Fung, Valentina Stosor, Nicholas Nissen, Michael T. Wong, Kenneth E. Sherman, Donald M. Stablein, Burc Barin – 9 December 2016 – Hepatitis C virus (HCV) infection is the leading cause of liver disease in hemophilia patients. In those with human immunodeficiency virus (HIV)/HCV coinfection, the rate of liver disease progression is greater than in HCV monoinfected individuals. Despite antiretroviral therapy, which slows HCV liver disease progression, some require transplantation.
Kojiro Taura, Toshimi Kaido, Takayuki Anazawa, Shintaro Yagi, Hideaki Okajima, Shinji Uemoto – 9 December 2016
Javier Brahm, Fernando Contreras – 5 December 2016