Treatment of hepatitis Delta
Heiner Wedemeyer, Svenja Hardtke, Michael P. Manns – 20 December 2013 – Watch a video presentation of this article
Heiner Wedemeyer, Svenja Hardtke, Michael P. Manns – 20 December 2013 – Watch a video presentation of this article
Rakesh Aggarwal – 20 December 2013 – Watch a video presentation of this article
Patrick J. Hackett, Marina Guirguis, Nozomi Sakai, Tetsuro Sakai – 19 December 2013 – Only 20.5% to 61.6% of abstracts presented at biomedical meetings are subsequently published as full‐length articles. The aim of this study was to analyze the abstract‐to‐publication rate of International Liver Transplantation Society (ILTS) meeting abstracts. Abstracts presented at 5 consecutive annual ILTS meetings (2004‐2008) were included to ensure a minimum follow‐up period of 4 years.
Mehlika Toy, Joshua A. Salomon, Hao Jiang, Honglian Gui, Hui Wang, Jiangshe Wang, Jan Hendrik Richardus, Qing Xie – 19 December 2013 – Inactive chronic hepatitis B (CHB) carriers make up the largest group of hepatitis B virus‐infected patients, and China bears the largest total CHB burden of any country. We therefore assessed the population health impact and cost‐effectiveness of a strategy of lifelong monitoring for inactive CHB and treatment of eligible patients in Shanghai, China.
Rolf Gebhardt – 19 December 2013
Long‐Bin Jeng, Ashok Thorat, Ping‐Chun Li, Ming‐Li Li, Horng‐Ren Yang, Chun‐Chieh Yeh, Te‐Hung Chen, Chia‐Hao Hsu, Shih‐Chao Hsu, Kin‐Shing Poon – 18 December 2013
Shigeru Kusumoto, Yasuhito Tanaka, Masashi Mizokami, Ryuzo Ueda – 17 December 2013
Alessandro Furlan, David Tulasne – 17 December 2013
Paul Martin, Andrea DiMartini, Sandy Feng, Robert Brown, Michael Fallon – 13 December 2013
Edward J. Andress, Michael Nicolaou, Marta R. Romero, Sandhia Naik, Peter H. Dixon, Catherine Williamson, Kenneth J. Linton – 12 December 2013 – ABCB4 flops phosphatidylcholine into the bile canaliculus to protect the biliary tree from the detergent activity of bile salts. Homozygous‐null ABCB4 mutations cause the childhood liver disease, progressive familial intrahepatic cholestasis, but cause and effect is less clear, with many missense mutations linked to less severe cholestatic diseases.