Nonadherence and the transition to adulthood
Emily M. Fredericks – 29 October 2009 – Key Points
Long‐term outcomes in pediatric liver transplantation
John Bucuvalas – 29 October 2009 – Key Points
HuR regulates gap junctional intercellular communication by controlling β‐catenin levels and adherens junction integrity
Niloofar Ale‐Agha, Stefanie Galban, Christiane Sobieroy, Kotb Abdelmohsen, Myriam Gorospe, Helmut Sies, Lars‐Oliver Klotz – 29 October 2009 – Gap junctional intercellular communication (GJIC) plays a critical role in the regulation of tissue homeostasis and carcinogenesis and is modulated by the levels, subcellular localization, and posttranslational modification of gap junction proteins, the connexins (Cx). Here, using oval cell‐like rat liver epithelial cells, we demonstrate that the RNA‐binding protein HuR promotes GJIC through two mechanisms.
Binge drinking–induced liver injury
Napoleon Waszkiewicz, Sławomir Dariusz Szajda, Anna Zalewska, Beata Konarzewska, Agata Szulc, Alina Kȩpka, Krzysztof Zwierz – 29 October 2009
Correction
29 October 2009
Autosomal dominant polycystic disease
Carlo Pedrolli, Emanuele Cereda – 29 October 2009
Medical hurricane: Health care reform and hepatology
Michael Charlton – 29 October 2009
Hepatitis B e antigen as a predictor for hepatitis B e antigen–positive chronic hepatitis B patients with peginterferon alfa‐2a therapy
En‐Qiang Chen, Hong Tang – 29 October 2009
Deletion of interleukin‐12p40 suppresses autoimmune cholangitis in dominant negative transforming growth factor β receptor type II mice
Katsunori Yoshida, Guo‐Xiang Yang, Weici Zhang, Masanobu Tsuda, Koichi Tsuneyama, Yuki Moritoki, Aftab A. Ansari, Kazuichi Okazaki, Zhe‐Xiong Lian, Ross L. Coppel, Ian R. Mackay, M.
Experimental models for hepatitis C viral infection
Andre Boonstra, Luc J. W. van der Laan, Thomas Vanwolleghem, Harry L. A. Janssen – 29 October 2009 – Hepatitis C virus (HCV) infection is a leading cause of chronic liver disease. The majority of infected individuals develop a persistent infection, which is associated with a high risk of liver cirrhosis and hepatocellular carcinoma. Since its discovery 20 years ago, progress in our understanding of this virus has been suboptimal due to the lack of good model systems.