Abstracts of the IASL biannual meeting
1 September 1988
1 September 1988
Sangkot Marzuki, Ian R. Mackay, Herawati Sudoyo, A. Saifuddin Noer – 1 September 1988
Maria Buti, Rafael Esteban, Michael Roggendorf, Juan Fernandez, Rosendo Jardi, Rudolf Rashofer, Helena Allende, Juan Genesca, Juan Ignacio Esteban, Jaime Guardia – 1 September 1988 – To evaluate the profile of hepatitis D virus replication and the corresponding immunoresponse after acute hepatitis D virus infection, sera from 50 patients with acute hepatitis D (36 with acute hepatitis B virus‐hepatitis D virus coinfection and 14 HBsAg carriers with hepatitis D virus superinfection) were investigated for the presence of hepatitis D virus RNA and other serological hepatitis D virus markers.
Marc I. Lorber – 1 September 1988
Kunio Okuda – 1 September 1988 – A new mutant causing hereditary hepatitis associated with severe jaundice has been discovered in the LEC strain of rats. Hepatitis appears suddenly in adult rats three to four months after birth. The clinical signs of hepatitis are characterized by severe jaundice, subcutaneous bleeding, oliguria, and loss of body weight. The affected rats showed a high lethality and histological changes of the liver with focal necrosis of enlarged hepatocytes without inflammatory cell response.
Neville L. Sandford, Ronald E. Saul – 1 September 1988 – Thirty‐six patients with advanced chronic liver disease of predominantly alcoholic etiology and with a documented history or current physical evidence of hepatic encephalopathy were studied and compared to 30 healthy controls. Assessment was made of their mental state, number connection test, venous blood ammonia, electroencephalography and visual evoked potentials with both pattern reversal and flash stimuli.
Roger F. Butterworth, Joël Lavoie, Jean‐François Giguère, Gilles Pomier‐Layrargues – 1 September 1988 – The integrity of GABA‐A receptors and of central benzodiazepine receptors was evaluated in membrane preparations from prefrontal cortex and caudate nuclei obtained at autopsy from nine cirrhotic patients who died in hepatic coma and an equal number of age‐matched control subjects. Histopathological studies revealed Alzheimer Type II astrocytosis in all cases in the cirrhotic group; controls were free from neurological, psychiatric or hepatic diseases.
Douglas M. Heuman, Z. Reno Vlahcevic, Marsha L. Bailey, Phillip B. Hylemon – 1 July 1988 – Bile acid synthesis is believed to be regulated by bile salts returning to the liver via the portal vein and suppressing cholesterol 7α‐hydroxylase, the rate‐limiting enzyme in the bile acid biosynthesis pathway. In order to characterize the relative effectiveness of bile salts in regulating bile acid synthesis, seven different bile acids were administered (1% w/w in chow) to rats over a 14‐day period.
Ann A. Jerkins, Robert D. Steele – 1 July 1988 – In a series of experiments, rats were subjected to end‐to‐side portacaval shunts using either suture or nonsuture surgical procedures. Rats were maintained on cereal‐based or purified diets in pellet form. All rats recovered preoperative body weights within the experimental periods; however, recovery of preoperative body weight was influenced by surgical technique and diet composition.