The effect of cirrhosis on radiogenomic biomarker's ability to predict microvascular invasion and outcome in hepatocellular carcinoma
Haiyun Tang, Harrison X. Bai, Chang Su, Ashley M. Lee, Li Yang – 26 April 2016
Cholangiocarcinoma risk factors and the potential role of aspirin
Fabio Bagante, T. Clark Gamblin, Timothy M. Pawlik – 26 April 2016
Hepatitis C management in prisons: An insight into daily clinical practice in three major Italian correctional houses
Antonella Foschi, Maddalena Casana, Anna Radice, Roberto Ranieri, Antonella d'Arminio Monforte – 26 April 2016
Intention‐to‐treat survival analysis of hepatitis C virus/human immunodeficiency virus coinfected liver transplant: Is it the waiting list?
Juan J. Araiz, M. Trinidad Serrano, Francisco A. García‐Gil, Elena M. Lacruz, Sara Lorente, José I. Sánchez, Miguel A. Suarez – 26 April 2016 – In human immunodeficiency virus (HIV)/hepatitis C virus (HCV) coinfected patients, the accelerated severity of liver disease, associated comorbidities, and mortality on the waiting list could change the possibility and results of liver transplantation (LT). Intention‐to‐treat survival analysis (ITTA) can accurately estimate the applicability and efficacy of LT.
Hepatitis C genotype 4R resistance‐associated polymorphisms: The achilles heel of the nonstructural 5A inhibitors?
Philippe Halfon, Sofiane Mohamed, Guillaume Penaranda, Hacène Khiri, Laurent Chiche, Carine Nicolas, Armand Abergel – 26 April 2016
Successful antiviral treatment of chronic hepatitis C leads to a rapid decline of liver stiffness without an early effect on spleen stiffness
Wim Verlinden, Sven Francque, Peter Michielsen, Thomas Vanwolleghem – 26 April 2016
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26 April 2016
Cell‐specific overactivation of nuclear erythroid 2 p45‐related factor 2–mediated gene expression in myeloid cells decreases hepatic ischemia/reperfusion injury
Lung‐Yi Lee, Calvin Harberg, Kristina A. Matkowskyj, Shelly Cook, Drew Roenneburg, Sabine Werner, Delinda A. Johnson, Jeffrey A. Johnson, David P. Foley – 26 April 2016 – Hepatic ischemia/reperfusion injury (IRI) is an unavoidable consequence of liver transplantation that can lead to postoperative hepatic dysfunction. Myeloid cells that include Kupffer cells, monocytes, and neutrophils contribute to the inflammatory response and cellular injury observed during hepatic IRI.