Maristela Pinheiro Freire, Ligia Câmera Pierrotti, Isabel Cristina Villela Soares Oshiro, Patrícia Rodrigues Bonazzi, Larissa Marques de Oliveira, Anna Silva Machado, Inneke Marie Van Der Heijden, Flavia Rossi, Silvia Figueiredo Costa, Luiz Augusto Carneiro D'Albuquerque, Edson Abdala – 19 December 2015 – Infection with carbapenem‐resistant Acinetobacter baumannii (CRAB) after liver transplantation (LT) is associated with high mortality.

Hui Chen, Fanny Turon, Virginia Hernández‐Gea, Josep Fuster, Angeles Garcia‐Criado, Marta Barrufet, Anna Darnell, Constantino Fondevila, Juan Carlos Garcia‐Valdecasas, Juan Carlos Garcia‐Pagán – 19 December 2015 – Portal vein thrombosis (PVT) occurs in approximately 2%‐26% of the patients awaiting liver transplantation (LT) and is no longer an absolute contraindication for LT. Nearly half of PVT cases are accidentally found during the LT procedure. The most important risk factor for PVT development in cirrhosis may be the severity of liver disease and reduced portal blood flow.

Jie Wei, Lei Fu, Shi‐fang Peng – 19 December 2015

Shigeru Marubashi, Hiroaki Nagano, Hidetoshi Eguchi, Hiroshi Wada, Tadafumi Asaoka, Yoshito Tomimaru, Akira Tomokuni, Koji Umeshita, Yuichiro Doki, Masaki Mori – 19 December 2015 – Small‐for‐size graft syndrome is an inevitable complication in living donor liver transplantation (LDLT). We hypothesized that graft weight (GW) measured after graft procurement is one of the variables predicting postoperative graft function. A total of 138 consecutive recipients of adult‐to‐adult LDLT between March 1999 and October 2014 were included in this study.

James P. Hamilton, Lahari Koganti, Abigael Muchenditsi, Venkata S. Pendyala, David Huso, Joseph Hankin, Robert C. Murphy, Dominik Huster, Uta Merle, Christopher Mangels, Nan Yang, James J. Potter, Esteban Mezey, Svetlana Lutsenko – 17 December 2015 – Wilson disease (WD) is a hepatoneurological disorder caused by mutations in the copper‐transporter, ATP7B. Copper accumulation in the liver is a hallmark of WD. Current therapy is based on copper chelation, which decreases the manifestations of liver disease, but often worsens neurological symptoms.

Kathie Béland, Fernando Alvarez – 17 December 2015

Matt Bohm, Raj Vuppalanchi, Naga Chalasani, The Drug‐Induced Liver Injury Network (DILIN) – 17 December 2015

James P. Hamilton, Lahari Koganti, Abigael Muchenditsi, Venkata S. Pendyala, David Huso, Joseph Hankin, Robert C. Murphy, Dominik Huster, Uta Merle, Christopher Mangels, Nan Yang, James J. Potter, Esteban Mezey, Svetlana Lutsenko – 17 December 2015 – Wilson disease (WD) is a hepatoneurological disorder caused by mutations in the copper‐transporter, ATP7B. Copper accumulation in the liver is a hallmark of WD. Current therapy is based on copper chelation, which decreases the manifestations of liver disease, but often worsens neurological symptoms.

Kunimaro Furuta, Yuichi Yoshida, Satoshi Ogura, Tomohide Kurahashi, Takashi Kizu, Shinichiro Maeda, Mayumi Egawa, Norihiro Chatani, Keigo Nishida, Yoshikazu Nakaoka, Shinichi Kiso, Yoshihiro Kamada, Tetsuo Takehara – 17 December 2015 – Acetaminophen (APAP) overdose is the leading cause of drug‐induced acute liver failure. In APAP‐induced acute liver failure, hepatocyte death and subsequent liver regeneration determines the prognosis of patients, making it necessary to identify suitable therapeutic targets based on detailed molecular mechanisms.

Mostafa M. Sira, Ahmad M. Sira – 17 December 2015