Patients with acute liver failure listed for superurgent liver transplantation in France: Reevaluation of the clichy‐villejuif criteria

Philippe Ichai, Camille Legeai, Claire Francoz, Karim Boudjema, Olivier Boillot, Christian Ducerf, Philippe Mathurin, François‐René Pruvot, Bertrand Suc, Philippe Wolf, Olivier Soubrane, Yves Patrice Le Treut, Daniel Cherqui, Laurent Hannoun, Georges‐Philippe Pageaux, Jean Gugenheim, Christian Letoublon, Jean Saric, Vincent Di Martino, Armand Abergel, Laurence Chiche, Teresa Maria Antonini, Christian Jacquelinet, Denis Castaing, Didier Samuel, the French Liver Transplant Teams – 11 February 2015 – In France, decisions regarding superurgent (SU) liver transplantation (LT) for patients with a

Implications of discordant findings between hepatic angiography and cross‐sectional imaging in transplant candidates with hepatocellular carcinoma

Kellie Young, Nicholas Fidelman, Francis Y. Yao, Nancy K. Hills, Maureen P. Kohi, K. Pallav Kolli, Andrew G. Taylor, Robert K. Kerlan – 11 February 2015 – The goal of this study was to determine whether the detection of discordant numbers of hypervascular foci at hepatic angiography versus contrast‐enhanced (CE) cross‐sectional imaging [computed tomography (CT) or magnetic resonance imaging (MRI)] is associated with adverse clinical outcomes in patients with hepatocellular carcinoma (HCC) who are listed for liver transplantation.

Delayed‐onset cytomegalovirus disease coded during hospital readmission in a multicenter, retrospective cohort of liver transplant recipients

Carlos A. Q. Santos, Daniel C. Brennan, William C. Chapman, Victoria J. Fraser, Margaret A. Olsen – 11 February 2015 – Delayed‐onset cytomegalovirus (CMV) disease can occur among liver transplant recipients after CMV prophylaxis is stopped. We hypothesized that delayed‐onset CMV disease (>100 days after transplant) occurs more commonly than early‐onset CMV disease and is associated with clinical sepsis and death.

Fibrates and cholestasis

Nisanne S. Ghonem, David N. Assis, James L. Boyer – 11 February 2015 – Cholestasis, including primary biliary cirrhosis (PBC) and primary sclerosing cholangitis (PSC), results from an impairment or disruption of bile production and causes intracellular retention of toxic bile constituents, including bile salts. If left untreated, cholestasis leads to liver fibrosis and cirrhosis, which eventually results in liver failure and the need for liver transplantation.

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