Hepatitis C virus promotes t‐helper (Th)17 responses through thymic stromal lymphopoietin production by infected hepatocytes
Hai‐Chon Lee, Sung‐Sang J. Sung, Peter D. Krueger, Yoon‐Ah Jo, Hugo R. Rosen, Steven F. Ziegler, Young S. Hahn – 13 November 2012 – Hepatitis C virus (HCV) is a major cause of liver cirrhosis and hepatocellular carcinoma. Here we report that infection of hepatic cells by HCV stimulates nuclear factor kappa B (NFκB)‐dependent production of thymic stromal lymphopoietin (TSLP). Hepatocyte‐derived TSLP in turn conditions dendritic cells (DCs) to drive T‐helper (Th)17 differentiation.