Treatment options for anti‐HCV treatment‐experienced patients
Peter Ferenci – 26 April 2012 – Watch the interview with the authors
Peter Ferenci – 26 April 2012 – Watch the interview with the authors
Diana Vetter, Michal Cohen‐Naftaly, Augusto Villanueva, Youngmin A. Lee, Peri Kocabayoglu, Rebekka Hannivoort, Goutham Narla, Josep M. Llovet, Swan N. Thung, Scott L. Friedman – 26 April 2012 – KLF6‐SV1 (SV1), the major splice variant of KLF6, antagonizes the KLF6 tumor suppressor by an unknown mechanism. Decreased KLF6 expression in human hepatocellular carcinoma (HCC) correlates with increased mortality, but the contribution of increased SV1 is unknown.
Jordan J. Feld – 26 April 2012 – Watch the interview with the authors
Douglas L. Nguyen, Timothy R. Morgan – 26 April 2012 – Watch the interview with the authors
Syed‐Mohammed R. Jafri, Stuart C. Gordon – 26 April 2012 – Watch the interview with the authors
Jean‐Michel Pawlotsky – 26 April 2012 – Watch the interview with the authors
Jean‐Michel Pawlotsky – 26 April 2012 – Watch the interview with the authors
M. Patrizia Carrieri, Philippe Sogni, Julien Cohen, Marc‐Arthur Loko, Maria Winnock, Bruno Spire, and the HEPAVIH Study Group – 26 April 2012
Johan M. Waern, Qinggong Yuan, Urda Rüdrich, Pablo D. Becker, Kai Schulze, Helene Strick‐Marchand, Nicholas D. Huntington, Behrend J. Zacher, Karsten Wursthorn, James P. DiSanto, Carlos A. Guzman, Michael P. Manns, Michael Ott, Michael Bock – 26 April 2012 – Macrophages play an important role in the rejection of xenogeneic cells and therefore represent a major obstacle to generating chimeric mice with human xenografts that are useful tools for basic and preclinical medical research.
Kiyoaki Ito, Atsushi Kuno, Yuzuru Ikehara, Masaya Sugiyama, Hiroaki Saito, Yoshihiko Aoki, Teppei Matsui, Masatoshi Imamura, Masaaki Korenaga, Kazumoto Murata, Naohiko Masaki, Yasuhito Tanaka, Shuhei Hige, Namiki Izumi, Masayuki Kurosaki, Shuhei Nishiguchi, Michiie Sakamoto, Masayoshi Kage, Hisashi Narimatsu, Masashi Mizokami – 26 April 2012 – Assessment of liver fibrosis in patients with chronic hepatitis C (CHC) is critical for predicting disease progression and determining future antiviral therapy.