Histological discrimination between autoimmune hepatitis and drug‐induced liver injury
Keiichi Fujiwara, Osamu Yokosuka – 31 October 2011
Keiichi Fujiwara, Osamu Yokosuka – 31 October 2011
Mirjam B. Zeisel, Daniel Da Costa, Thomas F. Baumert – 28 October 2011
Nicole A.W. Wood, May La Linn, David G. Bowen – 28 October 2011
Michael Charlton, Michael D. Leise – 28 October 2011
Mark D. Gorrell, Amany Zekry, Geoffrey W. McCaughan, Andrew Lloyd – 28 October 2011
Bruno Gridelli, Giovanni Vizzini, Giada Pietrosi, Angelo Luca, Marco Spada, Salvatore Gruttadauria, Davide Cintorino, Giandomenico Amico, Cinzia Chinnici, Toshio Miki, Eva Schmelzer, Pier Giulio Conaldi, Fabio Triolo, Jörg C. Gerlach – 27 October 2011 – Although hepatic cell transplantation (CT) holds the promise of bridging patients with end‐stage chronic liver failure to whole liver transplantation, suitable cell populations are under debate.
Alleluiah Rutebemberwa, Hugo R. Rosen – 27 October 2011
Georgios N. Kalambokis, Epameinondas V. Tsianos – 26 October 2011
Brigitte Grosse, Doris Cassio, Nadya Yousef, Céline Bernardo, Emmanuel Jacquemin, Emmanuel Gonzales – 26 October 2011 – Neonatal ichthyosis and sclerosing cholangitis (NISCH) syndrome is a liver disease caused by mutations of CLDN1 encoding Claudin‐1, a tight‐junction (TJ) protein. In this syndrome, it is speculated that cholestasis is caused by Claudin‐1 absence, leading to increased paracellular permeability and liver injuries secondary to paracellular bile regurgitation. We studied the role of claudin‐1 in hepatic paracellular permeability.
Ricky Harminder Bhogal, Barnaby Thomas Francis Stephenson, Simon Charles Afford – 26 October 2011