Activation of liver X receptor increases acetaminophen clearance and prevents its toxicity in mice
Simrat P.S. Saini, Bin Zhang, Yongdong Niu, Mengxi Jiang, Jie Gao, Yonggong Zhai, Jung Hoon Lee, Hirdesh Uppal, Hui Tian, Michael A. Tortorici, Samuel M. Poloyac, Wenxin Qin, Raman Venkataramanan, Wen Xie – 2 December 2011 – Overdose of acetaminophen (APAP), the active ingredient of Tylenol, is the leading cause of drug‐induced acute liver failure in the United States. As such, it is necessary to develop novel strategies to prevent or manage APAP toxicity. In this report, we reveal a novel function of the liver X receptor (LXR) in preventing APAP‐induced hepatotoxicity.