Dietary supplementation with methyl donor groups could prevent nonalcoholic fatty liver
Pául Cordero, Javier Campion, Fermín I. Milagro, J. Alfredo Martínez – 10 January 2011
Pául Cordero, Javier Campion, Fermín I. Milagro, J. Alfredo Martínez – 10 January 2011
Julian Schulze zur Wiesch, Neele Pudelski, Lena Hoepner, Michael Supplieth, Peter Buggisch, Ansgar W. Lohse, Stefan Lüth – 10 January 2011
Chyng‐Wen Fwu, Yin‐Chu Chien, San‐Lin You, Kenrad E. Nelson, Gregory D. Kirk, Hsu‐Sung Kuo, Manning Feinleib, Chien‐Jen Chen – 10 January 2011 – Few studies have evaluated the risk of cancers other than hepatocellular carcinoma associated with hepatitis B virus (HBV) infection. This study aimed to estimate incidence rates of intrahepatic cholangiocarcinoma (ICC) and non‐Hodgkin lymphoma (NHL) and its major subtypes in a nationwide cohort of parous women and to assess their associations with chronic HBV infection.
Feng Chen, Tomohide Hori, Norifumi Ohashi, Ann‐Marie Baine, Christopher B. Eckman, Justin H. Nguyen – 10 January 2011 – Mechanisms of brain edema in acute liver failure (ALF) are not completely understood. We recently demonstrated that matrix metalloproteinase 9 (MMP‐9) induces significant alterations to occludin in brain endothelial cells in vitro and in brains of mice with experimental ALF (Hepatology 2009;50:1914).
Hong‐Shiue Chou, Ching‐Chuan Hsieh, Horng‐Ren Yang, Lianfu Wang, Yusuke Arakawa, Kathleen Brown, Qingyu Wu, Feng Lin, Marion Peters, John J. Fung, Lina Lu, Shiguang Qian – 10 January 2011 – Although organ transplants have been applied for decades, outcomes of somatic cell transplants remain disappointing, presumably due to lack of appropriate supporting stromal cells.
Wen‐Horng Wang, Leo L. Studach, Ourania M. Andrisani – 10 January 2011 – Chronic hepatitis B virus (HBV) infection is a major etiologic factor in hepatocellular carcinoma (HCC) pathogenesis, involving effects of chronic liver inflammation and of the weakly oncogenic HBV X protein (pX). pX‐mediated hepatocyte transformation requires Polo‐like kinase1 (Plk1) activity, but the mechanism is not fully understood.
James F. Trotter, Brenda W. Gillespie, Norah A. Terrault, Michael M. Abecassis, Robert M. Merion, Robert S. Brown, Kim M. Olthoff, Paul H. Hayashi, Carl L. Berg, Robert A. Fisher, James E. Everhart, and the Adult‐to‐Adult Living Donor Liver Transplantation Cohort Study Group – 10 January 2011 – Information on the long‐term health of living liver donors is incomplete. Because changes in standard laboratory tests may reflect the underlying health of donors, results before and after donation were examined in the Adult‐to‐Adult Living Donor Liver Transplantation Cohort Study (A2ALL).
Sebastian Pratschke, Georgios Meimarakis, Stephan Mayr, Christian Graeb, Markus Rentsch, Reinhard Zachoval, Christiane Josephine Bruns, Axel Kleespies, Karl‐Walter Jauch, Florian Loehe, Martin Kurt Angele – 10 January 2011 – Proper liver perfusion is essential for sufficient organ function after liver transplantation. The aim of this study was to determine the effects of portal and arterial blood flow on liver function and organ survival after liver transplantation.
Vinay Sundaram, Deborah T. Jones, Nilesh H. Shah, Michael E. de Vera, Paulo Fontes, J. Wallis Marsh, Abhinav Humar, Jawad Ahmad – 10 January 2011 – Biliary complications remain a cause of morbidity after liver transplantation. The aim of this study was to determine whether changes in clinical practice in the era of the Model for End‐Stage Liver Disease (MELD) has affected biliary complications after liver transplantation. We retrospectively reviewed all deceased donor liver transplants at a single center.
Eishiro Mizukoshi, Yasunari Nakamoto, Kuniaki Arai, Tatsuya Yamashita, Akito Sakai, Yoshio Sakai, Takashi Kagaya, Taro Yamashita, Masao Honda, Shuichi Kaneko – 10 January 2011 – Many tumor‐associated antigens (TAAs) recognized by cytotoxic T cells (CTLs) have been identified during the last two decades and some of them have been used in clinical trials. However, there are very few in the field of immunotherapy for hepatocellular carcinoma (HCC) because there have not been comparative data regarding CTL responses to various TAAs.