Interleukin 18 binding protein ameliorates ischemia/reperfusion–induced hepatic injury in mice
Nikolaos Ouzounidis, Alexandros Giakoustidis, Theofilos Poutahidis, Katerina Angelopoulou, Stavros Iliadis, Antonis Chatzigiagkos, Argyro Zacharioudaki, Stamatis Angelopoulos, Apostolos Papalois, Vasilios Papanikolaou, Dimitrios Giakoustidis – 19 October 2015 – Inflammation‐associated oxidative stress contributes to hepatic ischemia/reperfusion injury (IRI). Detrimental inflammatory event cascades largely depend on activated Kupffer cells (KCs) and neutrophils, as well as proinflammatory cytokines, including tumor necrosis factor α (TNF‐α) and interleukin (IL) 18.