Tumor necrosis factor alpha‐induced receptor 1 signaling in alcoholic liver disease: A gut reaction?
Shirish Barve, Irina A. Kirpich, Craig. J. McClain – 6 December 2014
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Yeon Seok Seo, Chang Ho Jung, Tae Hyung Kim, Sun Young Yim, Soon Ho Um – 6 December 2014
Extracorporeal membrane oxygenation as a rescue device for postreperfusion cardiac arrest during liver transplantation
Margaret Tejani, Soo Youn Yi, Kyle W. Eudailey, Isaac George, James V. Guarrera, Gebhard Wagener – 6 December 2014
Liver transplantation in transthyretin amyloidosis: Issues and challenges
Andreia Carvalho, Ana Rocha, Luísa Lobato – 6 December 2014 – Hereditary transthyretin amyloidosis (ATTR) is a rare worldwide autosomal dominant disease caused by the systemic deposition of an amyloidogenic variant of transthyretin (TTR), which is usually derived from a single amino acid substitution in the TTR gene. More than 100 mutations have been described, with V30M being the most prevalent. Each variant has a different involvement, although peripheral neuropathy and cardiomyopathy are the most common.
IL‐22 and IL‐22 binding protein (IL‐22BP) regulate fibrosis and cirrhosis in hepatitis C virus and schistosome infections
Mathieu Sertorio, Xunya Hou, Rodrigo F.
Screening for occult hepatitis C virus infection: Does it need special attention?
Mohammad Saeid Rezaee‐Zavareh, Mahdi Ramezani‐Binabaj, Seyed Moayed Alavian – 5 December 2014
A sensor at the lipid‐protein interface: Lipid peroxidation controls hepatitis C virus replication
David Paul, Ralf Bartenschlager – 5 December 2014
Hepatitis C virus–induced reduction in miR‐181a impairs CD4+ T‐cell responses through overexpression of DUSP6
Guang Y. Li, Yun Zhou, Ruo S. Ying, Lei Shi, Yong Q. Cheng, Jun P. Ren, Jeddidiah W.D. Griffin, Zhan S. Jia, Chuan F. Li, Jonathan P. Moorman, Zhi Q. Yao – 5 December 2014 – T cells play a crucial role in viral clearance or persistence; however, the precise mechanisms that control their responses during viral infection remain incompletely understood. MicroRNA (miR) has been implicated as a key regulator controlling diverse biological processes through posttranscriptional repression.
A multi‐journal partnership to highlight joint first‐authors of manuscripts
M. Bishr Omary, Michael B. Wallace, Emad M. El‐Omar, Rajiv Jalan, Michael H. Nathanson – 5 December 2014
Autoimmune hepatitis in a murine autoimmune polyendocrine syndrome type 1 model is directed against multiple autoantigens
Matthias Hardtke‐Wolenski, Richard Taubert, Fatih Noyan, Maren Sievers, Janine Dywicki, Jerome Schlue, Christine S. Falk, Brita Ardesjö Lundgren, Hamish S. Scott, Andreas Pich, Mark S. Anderson, Michael P. Manns, Elmar Jaeckel – 5 December 2014 – Autoimmune polyendocrine syndrome type 1 (APS‐1) is caused by mutations of the autoimmune regulator (AIRE) gene. Mouse studies have shown that this results in defective negative selection of T cells and defective early seeding of peripheral organs with regulatory T cells (Tregs).