Budd‐chiari syndrome management: Timing of treatment is an open issue
Andrea Mancuso – 15 July 2013
Regulation of FOXO3 by phosphorylation and methylation in hepatitis C virus infection and alcohol exposure
Irina Tikhanovich, Sudhakiranmayi Kuravi, Roosevelt V. Campbell, Kusum K. Kharbanda, Antonio Artigues, Maria T. Villar, Steven A. Weinman – 15 July 2013 – Hepatitis C virus (HCV) infection produces chronic liver injury that is significantly exacerbated by alcohol consumption. While multiple mechanisms contribute to this synergy, a viral‐induced loss of antioxidant responses has been shown to play an important role.
Platelet count and survival of patients with compensated cirrhosis and small hepatocellular carcinoma treated with surgery
Edoardo G. Giannini, Vincenzo Savarino – 15 July 2013
A circuit of paracrine signals between liver sinusoid endothelial cells and hepatocytes regulates hepatitis C virus replication
Gisa Gerold, Thomas Pietschmann – 15 July 2013
Randomized, double‐blind, controlled study of glycerol phenylbutyrate in hepatic encephalopathy
Don C. Rockey, John M. Vierling, Parvez Mantry, Marwan Ghabril, Robert S. Brown, Olga Alexeeva, Igor A. Zupanets, Vladimir Grinevich, Andrey Baranovsky, Larysa Dudar, Galyna Fadieienko, Nataliya Kharchenko, Iryna Klaryts'ka, Vyacheslav Morozov, Priya Grewal, Timothy McCashland, K. Gautham Reddy, K. Rajender Reddy, Vasyl Syplyviy, Nathan M. Bass, Klara Dickinson, Catherine Norris, Dion Coakley, Masoud Mokhtarani, Bruce F.
Left ventricular dysfunction in obese children and adolescents with nonalcoholic fatty liver disease
Lucia Pacifico, Michele Di Martino, Antonio De Merulis, Mario Bezzi, John Frederick Osborn, Carlo Catalano, Claudio Chiesa – 11 July 2013 – Nonalcoholic fatty liver disease (NAFLD) may increase the risk for cardiac dysfunction. The present study aimed to determine whether, in children, NAFLD is associated with subclinical left ventricular (LV) structural and functional abnormalities independently of metabolic risk factors.
Left ventricular dysfunction in obese children and adolescents with nonalcoholic fatty liver disease
Lucia Pacifico, Michele Di Martino, Antonio De Merulis, Mario Bezzi, John Frederick Osborn, Carlo Catalano, Claudio Chiesa – 11 July 2013 – Nonalcoholic fatty liver disease (NAFLD) may increase the risk for cardiac dysfunction. The present study aimed to determine whether, in children, NAFLD is associated with subclinical left ventricular (LV) structural and functional abnormalities independently of metabolic risk factors.
Randomized, double‐blind, controlled study of glycerol phenylbutyrate in hepatic encephalopathy
Don C. Rockey, John M. Vierling, Parvez Mantry, Marwan Ghabril, Robert S. Brown, Olga Alexeeva, Igor A. Zupanets, Vladimir Grinevich, Andrey Baranovsky, Larysa Dudar, Galyna Fadieienko, Nataliya Kharchenko, Iryna Klaryts'ka, Vyacheslav Morozov, Priya Grewal, Timothy McCashland, K. Gautham Reddy, K. Rajender Reddy, Vasyl Syplyviy, Nathan M. Bass, Klara Dickinson, Catherine Norris, Dion Coakley, Masoud Mokhtarani, Bruce F.
Repopulation of the fibrotic/cirrhotic rat liver by transplanted hepatic stem/progenitor cells and mature hepatocytes
Mladen I. Yovchev, Yuhua Xue, David A. Shafritz, Joseph Locker, Michael Oertel – 10 July 2013 – Considerable progress has been made in developing antifibrotic agents and other strategies to treat liver fibrosis; however, significant long‐term restoration of functional liver mass has not yet been achieved. Therefore, we investigated whether transplanted hepatic stem/progenitor cells can effectively repopulate the liver with advanced fibrosis/cirrhosis.
Relationship between the histological appearance of the portal vein and development of ischemic‐type biliary lesions after liver transplantation
Waqar R. R. Farid, Jeroen Jonge, Pieter E. Zondervan, Ahmet Demirkiran, Herold J. Metselaar, Hugo W. Tilanus, Ron W. F. Bruin, Luc J. W. Laan, Geert Kazemier – 10 July 2013 – Ischemic‐type biliary lesions (ITBLs) are a major cause of morbidity after liver transplantation (LT). Their assumed underlying pathophysiological mechanism is ischemia/reperfusion injury of the biliary tree, in which the portal circulation has been proposed recently to have a role.