Patatin‐like phospholipase domain containing 3: A case in point linking genetic susceptibility for alcoholic and nonalcoholic liver disease
Devanshi Seth, Ann K. Daly, Paul S. Haber, Christopher P. Day – 29 March 2010
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Anna Ludovica Fracanzani, Alberto Piperno, Luca Valenti, Silvia Fargion – 26 March 2010
An aged T cell phenotype: A prognostic indicator in liver transplant recipients?
Janet M. Lord – 26 March 2010
The anti‐inflammatory effects of adiponectin are mediated via a heme oxygenase‐1–dependent pathway in rat Kupffer cells
Palash Mandal, Pil‐Hoon Park, Megan R. McMullen, Brian T. Pratt, Laura E. Nagy – 26 March 2010 – Altered expression and activity of immunomodulatory cytokines plays a major role in the pathogenesis of alcoholic liver disease. Chronic ethanol feeding increases the sensitivity of Kupffer cells, the resident hepatic macrophage, to lipopolysaccharide (LPS), leading to increased tumor necrosis factor alpha (TNF‐α) expression. This sensitization is normalized by treatment of primary cultures of Kupffer cells with adiponectin, an anti‐inflammatory adipokine.
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Justin H. Nguyen – 26 March 2010
A missing link in the puzzle: Brown adipose tissue
Tugrul Purnak, Ersan Ozaslan, Cumali Efe, Hasan Sevimler – 26 March 2010
Madelung's disease and alcoholic liver disorder
Mei‐Ju Ko, Hsien‐Ching Chiu – 26 March 2010
Now there are many (stages) where before there was one: In search of a pathophysiological classification of cirrhosis
Guadalupe Garcia‐Tsao, Scott Friedman, John Iredale, Massimo Pinzani – 26 March 2010
Hepatic resection versus radiofrequency ablation for very early stage hepatocellular carcinoma: A Markov model analysis
Yun Ku Cho, Jae Kyun Kim, Wan Tae Kim, Jin Wook Chung – 26 March 2010 – No adequate randomized trials have been reported for a comparison between hepatic resection (HR) versus radiofrequency ablation (RFA) for the treatment of patients with very early stage hepatocellular carcinoma (HCC), defined as an asymptomatic solitary HCC <2 cm.
Sorafenib inhibits the shedding of major histocompatibility complex class I–related chain A on hepatocellular carcinoma cells by down‐regulating a disintegrin and metalloproteinase 9
Keisuke Kohga, Tetsuo Takehara, Tomohide Tatsumi, Hisashi Ishida, Takuya Miyagi, Atsushi Hosui, Norio Hayashi – 26 March 2010 – The ectodomain of major histocompatibility complex class I–related chain A (MICA) is shed from tumor cells, and may be an important means of evading antitumor immunity. This study investigated the roles of a disintegrin and metalloproteinase 9 (ADAM9) in the shedding of MICA in human hepatocellular carcinoma (HCC).