Interleukin‐22 induces hepatic stellate cell senescence and restricts liver fibrosis in mice
Xiaoni Kong, Dechun Feng, Hua Wang, Feng Hong, Adeline Bertola, Fu‐Sheng Wang, Bin Gao – 2 April 2012 – Interleukin (IL)‐22 is known to play a key role in promoting antimicrobial immunity, inflammation, and tissue repair at barrier surfaces by binding to the receptors, IL‐10R2 and IL‐22R1. IL‐22R1 is generally thought to be expressed exclusively in epithelial cells. In this study, we identified high levels of IL‐10R2 and IL‐22R1 expression on hepatic stellate cells (HSCs), the predominant cell type involved in liver fibrogenesis in response to liver damage.