Hepatic endothelin‐1 and endocannabinoids‐dependent effects of hyperleptinemia in nonalcoholic steatohepatitis‐cirrhotic rats
Ying‐Ying Yang, Tung‐Hu Tsai, Yi‐Tsau Huang, Tzung‐Yan Lee, Che‐Chang Chan, Kuei‐Chuan Lee, Han‐Chieh Lin – 20 December 2011 – Leptin, the ob gene product, is a protein released from adipocytes and has been detected in fibrotic and cirrhotic livers. Leptin in brain has an inhibitory effect on food intake. Nonalcoholic steatohepatitis (NASH) is characterized by hyperleptinemia. This study explores the possible mechanisms of hyperleptinemia in relation to increased intrahepatic resistance (IHR) and portal hypertension in NASH cirrhotic rats.