Obesity, hyperlipidemia, and metabolic syndrome
Michael Charlton – 29 October 2009 – Key Points
What does the long‐term liver allograft look like for the pediatric recipient?
Stefan Hübscher – 29 October 2009 – Key Points
Quantification of genotype 4 hepatitis C virus RNA by the COBAS AmpliPrep/COBAS TaqMan hepatitis C virus test
Jeffrey J. Germer, Carl E. Bommersbach, Dana M. Schmidt, Jordan L. Bendel, Joseph D. C. Yao – 29 October 2009
Myeloperoxidase and superoxide dismutase 2 polymorphisms comodulate the risk of hepatocellular carcinoma and death in alcoholic cirrhosis
Pierre Nahon, Angela Sutton, Pierre Rufat, Marianne Ziol, Hassan Akouche, Christelle Laguillier, Nathalie Charnaux, Nathalie Ganne‐Carrié, Véronique Grando‐Lemaire, Gisèle N'Kontchou, Jean‐Claude Trinchet, Liliane Gattegno, Dominique Pessayre, Michel Beaugrand – 29 October 2009 – Alcohol increases reactive oxygen species (ROS) formation in hepatocyte mitochondria and by reduced nicotinamide adenine dinucleotide phosphate oxidases and myeloperoxidase (MPO) in Kupffer cells and liver‐infiltrating neutrophils.
Calpain activation by hepatitis C virus proteins inhibits the extrinsic apoptotic signaling pathway
Yannick Simonin, Olivier Disson, Hervé Lerat, Etienne Antoine, Fabien Binamé, Arielle R. Rosenberg, Solange Desagher, Patrice Lassus, Paulette Bioulac‐Sage, Urszula Hibner – 29 October 2009 – An unresolved question regarding the physiopathology of hepatitis C virus (HCV) infection is the remarkable efficiency with which host defenses are neutralized to establish chronic infection. Modulation of an apoptotic response is one strategy used by viruses to escape immune surveillance.
Long‐term outcomes of liver transplantation: Diabetes mellitus
Georges‐Philippe Pageaux, Stéphanie Faure, Hassan Bouyabrine, Michael Bismuth, Eric Assenat – 29 October 2009 – Key Points
Zinc treatment for symptomatic Wilson disease: Moving forward by looking back
Michael Schilsky – 29 October 2009
Coffee intake and chronic hepatitis C
Tugrul Purnak, Ersan Ozaslan – 29 October 2009
Modulation of glycosphingolipid metabolism significantly improves hepatic insulin sensitivity and reverses hepatic steatosis in mice
Nora Bijl, Milka Sokolović, Carlos Vrins, Mirjam Langeveld, Perry D. Moerland, Roelof Ottenhoff, Cindy P. A. A. van Roomen, Nike Claessen, Rolf G. Boot, Jan Aten, Albert K. Groen, Johannes M. F. G. Aerts, Marco van Eijk – 29 October 2009 – Nonalcoholic fatty liver disease (NAFLD) is associated with obesity, insulin resistance, and type 2 diabetes. The hyperinsulinemia that occurs as a consequence of insulin resistance is thought to be an important contributor to the development of fatty liver.