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Hossein Poutschi, Jacob George – 19 January 2012
Gall bladder polyps in primary sclerosing cholangitis: Adhere to the guidelines
Gregory Gores, Keith D. Lindor, Nataliya Razumilava – 19 January 2012
Mucin‐producing cholangiocarcinoma might derive from biliary tree stem/progenitor cells located in peribiliary glands
Vincenzo Cardinale, Yunfang Wang, Guido Carpino, Lola M. Reid, Eugenio Gaudio, Domenico Alvaro – 19 January 2012
SOFA score and acetaminophen‐induced acute liver failure
Darren G. Craig, Kenneth J. Simpson – 19 January 2012
Informed consent in surveillance for hepatocellular carcinoma in patients with cirrhosis
Ian A. Rowe, Matthew J. Armstrong, Richard Parker, Diarmaid D. Houlihan – 19 January 2012
Serum and glucocorticoid kinase 3 at 8q13.1 promotes cell proliferation and survival in hepatocellular carcinoma
Ming Liu, Leilei Chen, Tim Hon Man Chan, Jian Wang, Yan Li, Yan Li, Ting‐Ting Zeng, Yun‐Fei Yuan, Xin‐Yuan Guan – 19 January 2012 – Amplification of broad regions of 8q is one of the most frequent genetic alterations in hepatocellular carcinoma (HCC), suggesting the existence of oncogenes in addition to MYC at 8q24.21. In this report we examine the potential role of the candidate amplified oncogene serum and glucocorticoid kinase 3 (SGK3) at 8q13.1 in HCC pathogenesis.
Can we predict allograft tolerance in experimental animal models of transplantation?
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María Carlota Londoño, Eliano Bonaccorsi‐Riani, Alberto Sánchez‐Fueyo – 17 January 2012
Standard hepatic vein reconstruction with patch plasty using the native portal vein in adult living donor liver transplantation
Akira Mori, Toshimi Kaido, Yasuhiro Ogura, Kohei Ogawa, Koichiro Hata, Shintaro Yagi, Atsushi Yoshizawa, Hiroyoshi Isoda, Toshiya Shibata, Shinji Uemoto – 17 January 2012 – An outflow obstruction of the hepatic vein is a critical complication after living donor liver transplantation (LDLT) and occasionally leads to hepatic failure. Here we introduce a simple method for preventing outflow obstructions by patch plasty in adult LDLT. Between September 2001 and May 2010, 468 adult LDLT procedures were performed at Kyoto University Hospital.
Impact of the etiology of acute kidney injury on outcomes following liver transplantation: acute tubular necrosis versus hepatorenal syndrome
Mitra K. Nadim, Yuri S. Genyk, Chris Tokin, Jenny Fieber, Wanwarat Ananthapanyasut, Wei Ye, Rick Selby – 17 January 2012 – Acute kidney injury (AKI) at the time of liver transplantation (LT) has been associated with increased morbidity and mortality. In patients with potentially reversible renal dysfunction, predicting whether there will be sufficient return of native kidney function is sometimes difficult. Previous studies have focused mainly on the effect of the severity of renal dysfunction or the duration of pretransplant dialysis on posttransplant outcomes.