Mucin‐producing cholangiocarcinoma might derive from biliary tree stem/progenitor cells located in peribiliary glands
Vincenzo Cardinale, Yunfang Wang, Guido Carpino, Lola M. Reid, Eugenio Gaudio, Domenico Alvaro – 19 January 2012
Gall bladder polyps in primary sclerosing cholangitis: Adhere to the guidelines
Gregory Gores, Keith D. Lindor, Nataliya Razumilava – 19 January 2012
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Hossein Poutschi, Jacob George – 19 January 2012
Gallbladder polyps in primary sclerosing cholangitis: Indication for early intervention
Christoph Schramm, Ansgar W. Lohse – 19 January 2012
Cystic and papillary neoplasm involving peribiliary glands: A biliary counterpart of branch‐type intraductal papillary mucinous cystic neoplasm?
Yasuni Nakanuma, Yasunori Sato – 19 January 2012
AP2 adaptor complex mediates bile salt export pump internalization and modulates its hepatocanalicular expression and transport function
Hisamitsu Hayashi, Kaori Inamura, Kensuke Aida, Sotaro Naoi, Reiko Horikawa, Hironori Nagasaka, Tomozumi Takatani, Tamio Fukushima, Asami Hattori, Takashi Yabuki, Ikuo Horii, Yuichi Sugiyama – 19 January 2012 – The bile salt export pump (BSEP) mediates the biliary excretion of bile salts and its dysfunction induces intrahepatic cholestasis. Reduced canalicular expression of BSEP resulting from the promotion of its internalization is one of the causes of this disease state. However, the molecular mechanism underlying BSEP internalization from the canalicular membrane (CM) remains unknown.
Liver transplantation in septuagenarians receiving model for end‐stage liver disease exception points for hepatocellular carcinoma: The national experience
Jason J. Schwartz, Lisa Pappas, Heather F. Thiesset, Gabriela Vargas, John B. Sorensen, Robin D. Kim, William R. Hutson, Kenneth Boucher, Terry Box – 17 January 2012 – Current liver allocation policy in the United States grants liver transplant candidates with stage T2 hepatocellular carcinoma (HCC) a priority Model for End‐Stage Liver Disease (MELD) score of 22, regardless of age.
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Rafael San‐Juan, Jose Maria Aguado – 17 January 2012
Impact of the etiology of acute kidney injury on outcomes following liver transplantation: acute tubular necrosis versus hepatorenal syndrome
Mitra K. Nadim, Yuri S. Genyk, Chris Tokin, Jenny Fieber, Wanwarat Ananthapanyasut, Wei Ye, Rick Selby – 17 January 2012 – Acute kidney injury (AKI) at the time of liver transplantation (LT) has been associated with increased morbidity and mortality. In patients with potentially reversible renal dysfunction, predicting whether there will be sufficient return of native kidney function is sometimes difficult. Previous studies have focused mainly on the effect of the severity of renal dysfunction or the duration of pretransplant dialysis on posttransplant outcomes.
Standard hepatic vein reconstruction with patch plasty using the native portal vein in adult living donor liver transplantation
Akira Mori, Toshimi Kaido, Yasuhiro Ogura, Kohei Ogawa, Koichiro Hata, Shintaro Yagi, Atsushi Yoshizawa, Hiroyoshi Isoda, Toshiya Shibata, Shinji Uemoto – 17 January 2012 – An outflow obstruction of the hepatic vein is a critical complication after living donor liver transplantation (LDLT) and occasionally leads to hepatic failure. Here we introduce a simple method for preventing outflow obstructions by patch plasty in adult LDLT. Between September 2001 and May 2010, 468 adult LDLT procedures were performed at Kyoto University Hospital.